Published on Jul 11, 2018
I wanna give you the breakdown and a study guide to help you understand what probiotics you should really be looking for. It’s been a popular question that’s been coming up and I wanna address it and give you the signs and give you the research, and one of the probiotic strains I’m gonna talk about today is the most researched probiotic strain that exists to date. With over 1,000 studies backing it up in over 30 years of research. So I’m gonna talk about that one and I’m also gonna talk about another one that is extremely, extremely popular. Now I’m not talking about brands, I’m talking about strains. This isn’t any kind of pitch or anything, this is literally talking about the different stains of probiotic and you can find them on any label. So it’s gonna help you understand what you should be looking for, but also understanding the the general harmony of the gut biome and how it effects our immune system.
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Okay, so let’s start with L. rhamnosus. Lol what? Yeah, L. rhamnosus is the most popular, most researched strain of probiotic that is out there, like I said, with over 1,000 studies behind it and over 30 years of research. So L. rhamnosus is a very specific type of lactobacillus, and it’s been shown to do a lot of different things within the body, but in order to understand what Lactobacillus rhamnosus does, I wanna explain to you a very important cardinal rule, a cardinal educational foundation when it comes down to understanding probiotics and the gut biome in general. It’s called competitive adhesion.
You see, whenever anything bad passes through your body, we’re talking about a mold, we’re talking about a pathogen, bad bacteria, whatever, you name it, as it passes through your body and passes through your intestinal track, passes through your digestive system, it has to latch onto a cell before it can ever cause a problem. So, in an unideal world what’ll happen is that mold or that pathogen will attach to the cell and then it will trigger macrophages, or white blood cells, to come on and do their trick and do their whole thing, trigger inflammation, and you get sick, you go through the ropes, blah, blah, blah, blah, blah. Okay, but if we have a situation where a bad pathogen doesn’t have the ability to latch on to the cell in the first place, we don’t get sick, we don’t have an immune response. So how is this possible? Sounds like it’s way to good to be true.
Well when we have good bacteria in the gut, what ends up happening is these good bacteria will latch on to the cell for us. See what we have to remember is good bacteria is still a foreign bacteria, it’s still another living being inside of us as a living being. So although it’s supposed to be there, it’s kind of not supposed to be there. It’s sort of like kinda those birds that sit on top of a rhino or a hippo. It’s like their not born with those birds on them, they also play a critical role in the survival of the hippo or the rhino. So what ends up happening is the good bacteria latch onto the cell and the occupy the spaces where the bad bacteria normally would try to bind. So that’s exactly why, if you have more good bacteria than bad bacteria, you’re in a situation where the good bacteria will always prevent bad bacteria from making you sick. So that is exactly why they say that you’re immune system starts in your gut, and if you’re constantly using hand sanitizers or you’re constantly heating foods that destroy your gut bacteria, anyway, I digress, that’s gonna make it a big problem, right?
I’ll see you next time,
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